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{{tp|p=35073578|t=2022. Sulfated Progesterone Metabolites That Enhance Insulin Secretion via TRPM3 Are Reduced in Serum From Women With Gestational Diabetes Mellitus.|pdf=|usr=}}{{35073578}} {{tp|p=28122715|t=2017. Primary cilia regulate the osmotic stress response of renal epithelial cells through TRPM3.|pdf=|usr=}}{{28122715}} {{tp|p=32880575|t=2020. Upregulation of TRPM3 in nociceptors innervating inflamed tissue.|pdf=|usr=}}{{32880575}} {{tp|p=31736966|t=2019. Naltrexone Restores Impaired Transient Receptor Potential Melastatin 3 Ion Channel Function in Natural Killer Cells From Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Patients.|pdf=|usr=}}{{31736966}} {{tp|p=27727448|t=2017. Impaired calcium mobilization in natural killer cells from chronic fatigue syndrome/myalgic encephalomyelitis patients is associated with transient receptor potential melastatin 3 ion channels.|pdf=|usr=}}{{27727448}} {{tp|p=25650413|t=2015. Voriconazole, an antifungal triazol that causes visual side effects, is an inhibitor of TRPM1 and TRPM3 channels.|pdf=|usr=}}{{25650413}} {{tp|p=35172836|t=2022. Impaired TRPM3-dependent calcium influx and restoration using Naltrexone in natural killer cells of myalgic encephalomyelitis/chronic fatigue syndrome patients.|pdf=|usr=}}{{35172836}} {{tp|p=34266470|t=2021. The effect of IL-2 stimulation and treatment of TRPM3 on channel co-localisation with PIP(2) and NK cell function in myalgic encephalomyelitis/chronic fatigue syndrome patients.|pdf=|usr=}}{{34266470}} {{tp|p=31014226|t=2019. Validation of impaired Transient Receptor Potential Melastatin 3 ion channel activity in natural killer cells from Chronic Fatigue Syndrome/ Myalgic Encephalomyelitis patients.|pdf=|usr=}}{{31014226}} {{tp|p=30883612|t=2019. The TRPP2-dependent channel of renal primary cilia also requires TRPM3.|pdf=|usr=}}{{30883612}}
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